| Phenotype | Fish | Conditions | Figures | 
    
    
        
            | whole organism decreased length, abnormal | AB + MO1-igf2a | standard conditions | Fig. 1  from Hartnett et al., 2010 
 | 
    
        
            | somite morphology, abnormal | AB + MO1-igf2a | standard conditions | Fig. 1  from Hartnett et al., 2010 
 | 
    
        
            | vasculature increased accumulation blood, abnormal | AB + MO1-igf2a | standard conditions | Fig. S6  from Hartnett et al., 2010 
 | 
    
        
            | post-vent vasculature increased accumulation blood, abnormal | AB + MO1-igf2a | standard conditions | Fig. S6  from Hartnett et al., 2010 
 | 
    
        
            | blood decreased fluid flow, abnormal | AB + MO1-igf2a | standard conditions | Fig. S6  from Hartnett et al., 2010 
 | 
    
        
            | blood circulation disrupted, abnormal | AB + MO1-igf2a | standard conditions | Fig. 6  , 
                    
                        Fig. S6  from Hartnett et al., 2010 
 | 
    
        
            | heart increased size, abnormal | AB + MO1-igf2a | standard conditions | Fig. 6  from Hartnett et al., 2010 
 | 
    
        
            | eye decreased size, abnormal | AB + MO1-igf2a | standard conditions | Fig. 1  from Hartnett et al., 2010 
 | 
    
        
            | spinal cord neural keel apoptotic, abnormal | AB + MO1-igf2a | standard conditions | Fig. S4  from Hartnett et al., 2010 
 | 
    
        
            | brain structure, abnormal | AB + MO1-igf2a | standard conditions | Fig. 1  from Hartnett et al., 2010 
 | 
    
        
            | apoptotic process increased occurrence, abnormal | AB + MO1-igf2a | standard conditions | Fig. S4  from Hartnett et al., 2010 
 | 
    
        
            | intermediate cell mass of mesoderm distended, abnormal | AB + MO1-igf2a | standard conditions | Fig. 1  from Hartnett et al., 2010 
 | 
    
        
            | dorsal/ventral pattern formation disrupted, abnormal | AB + MO1-igf2a | standard conditions | Fig. S5  from Hartnett et al., 2010 
 | 
    
        
            | heart looping arrested, abnormal | AB + MO1-igf2a | standard conditions | Fig. 6  from Hartnett et al., 2010 
 | 
    
        
            | bulbus arteriosus morphology, abnormal | AB + MO1-igf2a | standard conditions | Fig. 6  from Hartnett et al., 2010 
 | 
    
        
            | trunk vasculature increased accumulation blood, abnormal | AB + MO1-igf2a | standard conditions | Fig. S6  from Hartnett et al., 2010 
 | 
    
        
            | whole organism wholly ventralized, abnormal | AB + MO1-igf2a | standard conditions | Fig. 1  , 
                    
                        Fig. S5  from Hartnett et al., 2010 
 | 
    
        
            | dorsal/ventral axis specification disrupted, abnormal | AB/TU + MO1-igf2a | standard conditions | Fig. 3
                    
                    from White et al., 2009 
 | 
    
        
            | notochord undulate, abnormal | AB/TU + MO1-igf2a | standard conditions | Fig. 3
                    
                    from White et al., 2009 
 | 
    
        
            | convergent extension involved in axis elongation delayed, abnormal | AB/TU + MO1-igf2a | standard conditions | Fig. 3
                    
                    from White et al., 2009 
 | 
    
        
            | whole organism curved ventral, abnormal | AB/TU + MO1-igf2a | standard conditions | Fig. 3
                    
                    from White et al., 2009 
 | 
    
        
            | notochord development delayed, abnormal | AB/TU + MO1-igf2a + MO2-igf2a + MO4-tp53 | standard conditions | Fig. 4
                    
                    from White et al., 2009 
 | 
    
        
            | somite specification disrupted, abnormal | AB/TU + MO1-igf2a + MO2-igf2a + MO4-tp53 | standard conditions | Fig. 4
                    
                    from White et al., 2009 
 | 
    
        
            | notochord undulate, abnormal | AB/TU + MO1-igf2a + MO2-igf2a + MO4-tp53 | standard conditions | Fig. 4
                    
                    from White et al., 2009 
 | 
    
        
            | trunk has fewer parts of type somite, abnormal | AB/TU + MO1-igf2a + MO2-igf2a + MO4-tp53 | standard conditions | Fig. 4
                    
                    from White et al., 2009 
 | 
    
        
            | angiogenesis disrupted, abnormal | y1Tg + MO1-igf2a | standard conditions | text only
                    
                    from Hartnett et al., 2010 
 | 
    
        
            | intersegmental vessel sprouting angiogenesis delayed, abnormal | y7Tg + MO1-igf2a + MO2-igf2a + MO4-tp53 | standard conditions | Fig. 4  from Dallinga et al., 2018 
 | 
    
        
            | intersegmental vessel filopodium absent, abnormal | y7Tg + MO1-igf2a + MO2-igf2a + MO4-tp53 | standard conditions | Fig. 4  from Dallinga et al., 2018 
 | 
    
        
            | blood circulation disrupted, abnormal | AB + MO1-igf2a + MO3-igf2b | standard conditions | Fig. 4  , 
                    
                        Fig. 6  from Hartnett et al., 2010 
 | 
    
        
            | blood decreased fluid flow, abnormal | AB + MO1-igf2a + MO3-igf2b | standard conditions | Fig. 4  from Hartnett et al., 2010 
 | 
    
        
            | dorsal/ventral pattern formation disrupted, abnormal | AB + MO1-igf2a + MO3-igf2b | standard conditions | Fig. 3  from Hartnett et al., 2010 
 | 
    
        
            | heart looping arrested, abnormal | AB + MO1-igf2a + MO3-igf2b | standard conditions | Fig. 6  from Hartnett et al., 2010 
 | 
    
        
            | whole organism decreased length, abnormal | AB + MO1-igf2a + MO3-igf2b | standard conditions | Fig. 1  from Hartnett et al., 2010 
 | 
    
        
            | brain structure, abnormal | AB + MO1-igf2a + MO3-igf2b | standard conditions | Fig. 1  from Hartnett et al., 2010 
 | 
    
        
            | vasculature increased accumulation blood, abnormal | AB + MO1-igf2a + MO3-igf2b | standard conditions | Fig. 4  from Hartnett et al., 2010 
 | 
    
        
            | intermediate cell mass of mesoderm distended, abnormal | AB + MO1-igf2a + MO3-igf2b | standard conditions | Fig. 1  from Hartnett et al., 2010 
 | 
    
        
            | somite morphology, abnormal | AB + MO1-igf2a + MO3-igf2b | standard conditions | Fig. 1  from Hartnett et al., 2010 
 | 
    
        
            | post-vent vasculature increased accumulation blood, abnormal | AB + MO1-igf2a + MO3-igf2b | standard conditions | Fig. 4  from Hartnett et al., 2010 
 | 
    
        
            | apoptotic process increased occurrence, abnormal | AB + MO1-igf2a + MO3-igf2b | standard conditions | Fig. 2  from Hartnett et al., 2010 
 | 
    
        
            | eye decreased size, abnormal | AB + MO1-igf2a + MO3-igf2b | standard conditions | Fig. 1  from Hartnett et al., 2010 
 | 
    
        
            | bulbus arteriosus morphology, abnormal | AB + MO1-igf2a + MO3-igf2b | standard conditions | Fig. 6  from Hartnett et al., 2010 
 | 
    
        
            | trunk vasculature increased accumulation blood, abnormal | AB + MO1-igf2a + MO3-igf2b | standard conditions | Fig. 4  from Hartnett et al., 2010 
 | 
    
        
            | heart increased size, abnormal | AB + MO1-igf2a + MO3-igf2b | standard conditions | Fig. 6  from Hartnett et al., 2010 
 | 
    
        
            | whole organism wholly ventralized, abnormal | AB + MO1-igf2a + MO3-igf2b | standard conditions | Fig. 1  , 
                    
                        Fig. 3  from Hartnett et al., 2010 
 | 
    
        
            | heart contraction decreased occurrence, abnormal | AB + MO1-igf2a + MO3-igf2b | standard conditions | text only
                    
                    from Hartnett et al., 2010 
 | 
    
        
            | neural keel apoptotic, abnormal | AB + MO1-igf2a + MO3-igf2b | standard conditions | Fig. 2  from Hartnett et al., 2010 
 | 
    
        
            | spinal cord neural keel apoptotic, abnormal | AB + MO1-igf2a + MO3-igf2b | standard conditions | Fig. 2  from Hartnett et al., 2010 
 | 
    
        
            | whole organism curved ventral, abnormal | AB/TU + MO1-igf2a + MO1-igf2b + MO2-igf2a + MO2-igf2b + MO4-tp53 | standard conditions | text only
                    
                    from White et al., 2009 
 | 
    
        
            | somite specification disrupted, abnormal | AB/TU + MO1-igf2a + MO1-igf2b + MO2-igf2a + MO2-igf2b + MO4-tp53 | standard conditions | Fig. 4
                    
                    from White et al., 2009 
 | 
    
        
            | notochord undulate, abnormal | AB/TU + MO1-igf2a + MO1-igf2b + MO2-igf2a + MO2-igf2b + MO4-tp53 | standard conditions | text only
                    
                    from White et al., 2009 
 | 
    
        
            | trunk has fewer parts of type somite, abnormal | AB/TU + MO1-igf2a + MO1-igf2b + MO2-igf2a + MO2-igf2b + MO4-tp53 | standard conditions | Fig. 4
                    
                    from White et al., 2009 
 | 
    
        
            | cranial blood vessel decreased size, abnormal | y1Tg + MO1-igf2a + MO3-igf2b | standard conditions | Fig. 5  from Hartnett et al., 2010 
 | 
    
        
            | angiogenesis disrupted, abnormal | y1Tg + MO1-igf2a + MO3-igf2b | standard conditions | Fig. 5  , 
                    
                        Fig. S7  , 
                    
                        text only
                    
                    from Hartnett et al., 2010 
 | 
    
        
            | parachordal vessel hypoplastic, abnormal | y1Tg + MO1-igf2a + MO3-igf2b | standard conditions | Fig. 5  from Hartnett et al., 2010 
 | 
    
        
            | intersegmental vessel decreased size, abnormal | y1Tg + MO1-igf2a + MO3-igf2b | standard conditions | Fig. 5  from Hartnett et al., 2010 
 | 
    
        
            | parachordal vessel morphology, abnormal | y1Tg + MO1-igf2a + MO3-igf2b | standard conditions | Fig. 5  from Hartnett et al., 2010 
 | 
    
        
            | intermediate cell mass of mesoderm distended, abnormal | y1Tg + MO1-igf2a + MO3-igf2b | standard conditions | Fig. 5  from Hartnett et al., 2010 
 | 
    
        
            | intersegmental vessel disorganized, abnormal | y1Tg + MO1-igf2a + MO3-igf2b | standard conditions | Fig. 5  from Hartnett et al., 2010 
 | 
    
        
            | vasculature disorganized, abnormal | y1Tg + MO1-igf2a + MO3-igf2b | standard conditions | Fig. S7  from Hartnett et al., 2010 
 | 
    
        
            | ocular blood vessel decreased size, abnormal | y1Tg + MO1-igf2a + MO3-igf2b | standard conditions | Fig. 5  from Hartnett et al., 2010 
 | 
    
        
            | sprouting angiogenesis decreased occurrence, abnormal | y1Tg + MO1-igf2a + MO3-igf2b | standard conditions | Fig. 5  from Hartnett et al., 2010 
 |