Morpholino

MO1-ap1g2

ID
ZDB-MRPHLNO-170808-6
Name
MO1-ap1g2
Previous Names
None
Target
Sequence
5' - GGTCACTGCAACACCAGCAGAGTAA - 3'
ZFIN BLAST NCBI BLAST Ensembl UCSC BLAT
Disclaimer
Although ZFIN verifies reagent sequence data, we recommend that you conduct independent sequence analysis before ordering any reagent.
Note
Splice-blocking MO.
Genome Resources
None
Target Location
Genomic Features
No data available
Expression
Gene expression in Wild Types + MO1-ap1g2
No data available
Phenotype
Phenotype resulting from MO1-ap1g2
Phenotype Fish Figures
artery malformed, abnormal sd2Tg; y1Tg + MO1-ap1g2 Fig. 7 from Zizioli et al., 2017
blood cell mislocalised, abnormal sd2Tg; y1Tg + MO1-ap1g2 Fig. 7 from Zizioli et al., 2017
blood circulation decreased occurrence, abnormal sd2Tg; y1Tg + MO1-ap1g2 Fig. 7 from Zizioli et al., 2017
blood vessel lumenization decreased occurrence, abnormal sd2Tg; y1Tg + MO1-ap1g2 Fig. 7 from Zizioli et al., 2017
caudal fin curved, abnormal WT + MO1-ap1g2 Fig. 5 from Zizioli et al., 2017
caudal fin kinked, abnormal WT + MO1-ap1g2 Fig. 5 from Zizioli et al., 2017
diencephalon neurog1 expression decreased amount, abnormal WT + MO1-ap1g2 Fig. 6 from Zizioli et al., 2017
dorsal longitudinal anastomotic vessel malformed, abnormal sd2Tg; y1Tg + MO1-ap1g2 Fig. 7 from Zizioli et al., 2017
forebrain neurod1 expression decreased amount, abnormal WT + MO1-ap1g2 Fig. 6 from Zizioli et al., 2017
forebrain morphology, abnormal WT + MO1-ap1g2 Fig. 6 from Zizioli et al., 2017
fourth ventricle absent, abnormal WT + MO1-ap1g2 Fig. 6 from Zizioli et al., 2017
fourth ventricle lef1 expression decreased amount, abnormal WT + MO1-ap1g2 Fig. 6 from Zizioli et al., 2017
head morphology, abnormal WT + MO1-ap1g2 Fig. 5 from Zizioli et al., 2017
heart edematous, abnormal WT + MO1-ap1g2 Fig. 5 from Zizioli et al., 2017
hindbrain neurog1 expression decreased amount, abnormal WT + MO1-ap1g2 Fig. 6 from Zizioli et al., 2017
hindbrain pax2a expression decreased amount, abnormal WT + MO1-ap1g2 Fig. 6 from Zizioli et al., 2017
hindbrain neurod1 expression decreased amount, abnormal WT + MO1-ap1g2 Fig. 6 from Zizioli et al., 2017
hindbrain morphology, abnormal WT + MO1-ap1g2 Fig. 6 from Zizioli et al., 2017
intersegmental vessel malformed, abnormal sd2Tg; y1Tg + MO1-ap1g2 Fig. 7 from Zizioli et al., 2017
midbrain neurod1 expression decreased amount, abnormal WT + MO1-ap1g2 Fig. 6 from Zizioli et al., 2017
midbrain morphology, abnormal WT + MO1-ap1g2 Fig. 6 from Zizioli et al., 2017
midbrain hindbrain boundary lef1 expression decreased amount, abnormal WT + MO1-ap1g2 Fig. 6 from Zizioli et al., 2017
midbrain hindbrain boundary pax2a expression decreased amount, abnormal WT + MO1-ap1g2 Fig. 6 from Zizioli et al., 2017
midbrain hindbrain boundary neurod1 expression decreased amount, abnormal WT + MO1-ap1g2 Fig. 6 from Zizioli et al., 2017
somite shape, abnormal WT + MO1-ap1g2 Fig. 5 from Zizioli et al., 2017
spinal cord neuron neurog1 expression decreased amount, abnormal WT + MO1-ap1g2 Fig. 6 from Zizioli et al., 2017
tegmentum neurog1 expression decreased amount, abnormal WT + MO1-ap1g2 Fig. 6 from Zizioli et al., 2017
telencephalon lef1 expression decreased amount, abnormal WT + MO1-ap1g2 Fig. 6 from Zizioli et al., 2017
trunk decreased thickness, abnormal WT + MO1-ap1g2 Fig. 5 from Zizioli et al., 2017
vein malformed, abnormal sd2Tg; y1Tg + MO1-ap1g2 Fig. 7 from Zizioli et al., 2017
ventral fin fold absent, abnormal WT + MO1-ap1g2 Fig. 5 from Zizioli et al., 2017
ventral fin fold decreased size, abnormal WT + MO1-ap1g2 Fig. 5 from Zizioli et al., 2017
Phenotype of all Fish created by or utilizing MO1-ap1g2
Phenotype Fish Conditions Figures
fourth ventricle lef1 expression decreased amount, abnormal WT + MO1-ap1g2 control Fig. 6 from Zizioli et al., 2017
telencephalon lef1 expression decreased amount, abnormal WT + MO1-ap1g2 control Fig. 6 from Zizioli et al., 2017
ventral fin fold decreased size, abnormal WT + MO1-ap1g2 control Fig. 5 from Zizioli et al., 2017
midbrain hindbrain boundary neurod1 expression decreased amount, abnormal WT + MO1-ap1g2 control Fig. 6 from Zizioli et al., 2017
caudal fin kinked, abnormal WT + MO1-ap1g2 control Fig. 5 from Zizioli et al., 2017
midbrain neurod1 expression decreased amount, abnormal WT + MO1-ap1g2 control Fig. 6 from Zizioli et al., 2017
caudal fin curved, abnormal WT + MO1-ap1g2 control Fig. 5 from Zizioli et al., 2017
diencephalon neurog1 expression decreased amount, abnormal WT + MO1-ap1g2 control Fig. 6 from Zizioli et al., 2017
tegmentum neurog1 expression decreased amount, abnormal WT + MO1-ap1g2 control Fig. 6 from Zizioli et al., 2017
fourth ventricle absent, abnormal WT + MO1-ap1g2 control Fig. 6 from Zizioli et al., 2017
hindbrain pax2a expression decreased amount, abnormal WT + MO1-ap1g2 control Fig. 6 from Zizioli et al., 2017
forebrain morphology, abnormal WT + MO1-ap1g2 control Fig. 6 from Zizioli et al., 2017
head morphology, abnormal WT + MO1-ap1g2 control Fig. 5 from Zizioli et al., 2017
spinal cord neuron neurog1 expression decreased amount, abnormal WT + MO1-ap1g2 control Fig. 6 from Zizioli et al., 2017
hindbrain morphology, abnormal WT + MO1-ap1g2 control Fig. 6 from Zizioli et al., 2017
midbrain hindbrain boundary pax2a expression decreased amount, abnormal WT + MO1-ap1g2 control Fig. 6 from Zizioli et al., 2017
forebrain neurod1 expression decreased amount, abnormal WT + MO1-ap1g2 control Fig. 6 from Zizioli et al., 2017
somite shape, abnormal WT + MO1-ap1g2 control Fig. 5 from Zizioli et al., 2017
heart edematous, abnormal WT + MO1-ap1g2 control Fig. 5 from Zizioli et al., 2017
hindbrain neurod1 expression decreased amount, abnormal WT + MO1-ap1g2 control Fig. 6 from Zizioli et al., 2017
ventral fin fold absent, abnormal WT + MO1-ap1g2 control Fig. 5 from Zizioli et al., 2017
midbrain hindbrain boundary lef1 expression decreased amount, abnormal WT + MO1-ap1g2 control Fig. 6 from Zizioli et al., 2017
trunk decreased thickness, abnormal WT + MO1-ap1g2 control Fig. 5 from Zizioli et al., 2017
midbrain morphology, abnormal WT + MO1-ap1g2 control Fig. 6 from Zizioli et al., 2017
hindbrain neurog1 expression decreased amount, abnormal WT + MO1-ap1g2 control Fig. 6 from Zizioli et al., 2017
intersegmental vessel malformed, abnormal sd2Tg; y1Tg + MO1-ap1g2 control Fig. 7 from Zizioli et al., 2017
blood cell mislocalised, abnormal sd2Tg; y1Tg + MO1-ap1g2 control Fig. 7 from Zizioli et al., 2017
dorsal longitudinal anastomotic vessel malformed, abnormal sd2Tg; y1Tg + MO1-ap1g2 control Fig. 7 from Zizioli et al., 2017
blood circulation decreased occurrence, abnormal sd2Tg; y1Tg + MO1-ap1g2 control Fig. 7 from Zizioli et al., 2017
artery malformed, abnormal sd2Tg; y1Tg + MO1-ap1g2 control Fig. 7 from Zizioli et al., 2017
blood vessel lumenization decreased occurrence, abnormal sd2Tg; y1Tg + MO1-ap1g2 control Fig. 7 from Zizioli et al., 2017
vein malformed, abnormal sd2Tg; y1Tg + MO1-ap1g2 control Fig. 7 from Zizioli et al., 2017
telencephalon lef1 expression decreased amount, abnormal WT + MO1-ap1g2 + MO2-ap1g1 control Fig. 6 from Zizioli et al., 2017
fourth ventricle lef1 expression decreased amount, abnormal WT + MO1-ap1g2 + MO2-ap1g1 control Fig. 6 from Zizioli et al., 2017
midbrain hindbrain boundary neurod1 expression decreased amount, abnormal WT + MO1-ap1g2 + MO2-ap1g1 control Fig. 6 from Zizioli et al., 2017
caudal fin kinked, abnormal WT + MO1-ap1g2 + MO2-ap1g1 control Fig. 5 from Zizioli et al., 2017
midbrain neurod1 expression decreased amount, abnormal WT + MO1-ap1g2 + MO2-ap1g1 control Fig. 6 from Zizioli et al., 2017
ventral fin fold decreased size, abnormal WT + MO1-ap1g2 + MO2-ap1g1 control Fig. 5 from Zizioli et al., 2017
caudal fin curved, abnormal WT + MO1-ap1g2 + MO2-ap1g1 control Fig. 5 from Zizioli et al., 2017
tegmentum neurog1 expression decreased amount, abnormal WT + MO1-ap1g2 + MO2-ap1g1 control Fig. 6 from Zizioli et al., 2017
fourth ventricle absent, abnormal WT + MO1-ap1g2 + MO2-ap1g1 control Fig. 6 from Zizioli et al., 2017
hindbrain pax2a expression decreased amount, abnormal WT + MO1-ap1g2 + MO2-ap1g1 control Fig. 6 from Zizioli et al., 2017
diencephalon neurog1 expression decreased amount, abnormal WT + MO1-ap1g2 + MO2-ap1g1 control Fig. 6 from Zizioli et al., 2017
forebrain morphology, abnormal WT + MO1-ap1g2 + MO2-ap1g1 control Fig. 6 from Zizioli et al., 2017
head morphology, abnormal WT + MO1-ap1g2 + MO2-ap1g1 control Fig. 5 from Zizioli et al., 2017
midbrain hindbrain boundary lef1 expression decreased amount, abnormal WT + MO1-ap1g2 + MO2-ap1g1 control Fig. 6 from Zizioli et al., 2017
whole organism decreased life span, abnormal WT + MO1-ap1g2 + MO2-ap1g1 control Fig. 5 from Zizioli et al., 2017
notochord axis elongation decreased occurrence, abnormal WT + MO1-ap1g2 + MO2-ap1g1 control Fig. 8 from Zizioli et al., 2017
hindbrain morphology, abnormal WT + MO1-ap1g2 + MO2-ap1g1 control Fig. 6 from Zizioli et al., 2017
forebrain neurod1 expression decreased amount, abnormal WT + MO1-ap1g2 + MO2-ap1g1 control Fig. 6 from Zizioli et al., 2017
midbrain hindbrain boundary pax2a expression decreased amount, abnormal WT + MO1-ap1g2 + MO2-ap1g1 control Fig. 6 from Zizioli et al., 2017
spinal cord neuron neurog1 expression decreased amount, abnormal WT + MO1-ap1g2 + MO2-ap1g1 control Fig. 6 from Zizioli et al., 2017
heart edematous, abnormal WT + MO1-ap1g2 + MO2-ap1g1 control Fig. 5 from Zizioli et al., 2017
somite shape, abnormal WT + MO1-ap1g2 + MO2-ap1g1 control Fig. 5 from Zizioli et al., 2017
hindbrain neurod1 expression decreased amount, abnormal WT + MO1-ap1g2 + MO2-ap1g1 control Fig. 6 from Zizioli et al., 2017
ventral fin fold absent, abnormal WT + MO1-ap1g2 + MO2-ap1g1 control Fig. 5 from Zizioli et al., 2017
trunk decreased thickness, abnormal WT + MO1-ap1g2 + MO2-ap1g1 control Fig. 5 from Zizioli et al., 2017
midbrain morphology, abnormal WT + MO1-ap1g2 + MO2-ap1g1 control Fig. 6 from Zizioli et al., 2017
hindbrain neurog1 expression decreased amount, abnormal WT + MO1-ap1g2 + MO2-ap1g1 control Fig. 6 from Zizioli et al., 2017
trunk EGFP expression decreased amount, abnormal mw29Tg + MO1-ap1g2 + MO2-ap1g1 control Fig. 8 from Zizioli et al., 2017
BMP signaling pathway decreased occurrence, abnormal mw29Tg + MO1-ap1g2 + MO2-ap1g1 control Fig. 8 from Zizioli et al., 2017
blood cell mislocalised, abnormal sd2Tg; y1Tg + MO1-ap1g2 + MO2-ap1g1 control Fig. 7 from Zizioli et al., 2017
intersegmental vessel malformed, abnormal sd2Tg; y1Tg + MO1-ap1g2 + MO2-ap1g1 control Fig. 7 from Zizioli et al., 2017
dorsal longitudinal anastomotic vessel malformed, abnormal sd2Tg; y1Tg + MO1-ap1g2 + MO2-ap1g1 control Fig. 7 from Zizioli et al., 2017
blood circulation decreased occurrence, abnormal sd2Tg; y1Tg + MO1-ap1g2 + MO2-ap1g1 control Fig. 7 from Zizioli et al., 2017
artery malformed, abnormal sd2Tg; y1Tg + MO1-ap1g2 + MO2-ap1g1 control Fig. 7 from Zizioli et al., 2017
vein malformed, abnormal sd2Tg; y1Tg + MO1-ap1g2 + MO2-ap1g1 control Fig. 7 from Zizioli et al., 2017
blood vessel lumenization decreased occurrence, abnormal sd2Tg; y1Tg + MO1-ap1g2 + MO2-ap1g1 control Fig. 7 from Zizioli et al., 2017
Citations
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