Publication	Data		Fish	Anatomy
Toms et al., 2024	FIGURE 2		AB + MO5-ntn1a	macula stereocilium bundle decreased amount, abnormal neuromast hair cell cell body absence of anatomical entity, abnormal neuromast hair cell kinocilium absence of anatomical entity, abnormal neuromast hair cell stereocilium bundle absence of anatomical entity, abnormal neuromast hair cell stereocilium bundle decreased amount, abnormal optic fissure closure incomplete, abnormal
Hardy et al., 2019	Figure 4 - figure supplement 3		ntn1anni1/nni1 ,   WT + MO5-ntn1a	closure of optic fissure decreased occurrence, abnormal eye decreased size, abnormal optic fissure closure incomplete, abnormal optic fissure open, abnormal
Li et al., 2019	Fig. 3		ntn1azf3231/zf3231	embryonic viscerocranium morphogenesis process quality, abnormal
Richardson et al., 2019	Fig. 7		AB + MO5-ntn1a	closure of optic fissure decreased occurrence, abnormal optic fissure closure incomplete, abnormal optic fissure morphology, abnormal optic fissure open, abnormal
	Fig. 8		AB + MO5-ntn1a	eye atoh7 expression decreased amount, abnormal
Opitz et al., 2015	Fig. 3		WT + MO2-ntn1a	heart jogging disrupted, abnormal heart looping disrupted, abnormal thyroid follicle position, abnormal thyroid primordium morphology, abnormal
	Fig. 4		s843Tg + MO2-ntn1a ,   s843Tg; ulb1Tg + MO2-ntn1a	aortic arch absent, abnormal aortic arch hypoplastic, abnormal aortic arch morphology, abnormal hypobranchial artery dysplastic, abnormal hypobranchial artery morphology, abnormal pharyngeal vasculature morphology, abnormal thyroid follicle mislocalised, abnormal thyroid primordium mislocalised, abnormal ventral aorta morphology, abnormal ventral aorta separated from bulbus arteriosus, abnormal
	Fig. 5		s843Tg; ulb1Tg + MO2-ntn1a	thyroid primordium distended, abnormal thyroid primordium mislocalised, abnormal thyroid primordium morphology, abnormal
	Fig. 6		ulb1Tg + MO2-ntn1a	thyroid follicle cell decreased amount, abnormal
	Fig. S1		s843Tg + MO2-ntn1a	parachordal vessel incomplete structure, abnormal parachordal vessel morphology, abnormal
Tu et al., 2015	Fig. 6		la116Tg + MO4-ntn1a ,   is5Tg; y7Tg + MO4-ntn1a	intersegmental vessel endothelial cell decreased amount, abnormal vasculature development disrupted, abnormal whole organism lacks all parts of type parachordal vessel, abnormal
	Fig. S6		is5Tg; y7Tg + MO4-ntn1a	blood vessel endothelial cell migration decreased process quality, abnormal blood vessel endothelial cell proliferation involved in sprouting angiogenesis decreased process quality, abnormal
Zhang et al., 2012	Fig. 7		b1204Tg + MO4-ntn1a	anterior commissure axon extension process quality, abnormal anterior commissure morphogenesis process quality, abnormal
Hale et al., 2011	Fig. 2		WT + MO2-ntn1a	VaP motor neuron axon increased branchiness, abnormal VaP motor neuron axon protruding into muscle pioneer, abnormal
Lim et al., 2011	Fig. 6		ml2Tg + MO2-ntn1a	horizontal myoseptum lacks parts or has fewer parts of type motor neuron axon, abnormal horizontal myoseptum lacks parts or has fewer parts of type RoP motor neuron axon, abnormal
	Fig. S1		y1Tg/+ + MO2-ntn1a	lymph vessel development process quality, abnormal thoracic duct aplastic, abnormal
Guenebeaud et al., 2010	Fig. 7		y1Tg + MO1-ntn1a	cysteine-type endopeptidase activator activity increased process quality, abnormal
Castets et al., 2009	Fig. 3		y1Tg + MO3-ntn1a ,   y1Tg + MO1-ntn1a	apoptotic process increased occurrence, abnormal embryo development disrupted, abnormal intersegmental vessel disorganized, abnormal whole organism lacks all parts of type dorsal longitudinal anastomotic vessel, abnormal whole organism lacks parts or has fewer parts of type intersegmental vessel, abnormal whole organism lacks parts or has fewer parts of type parachordal vessel, abnormal
Wilson et al., 2006	Fig. 3		y1Tg + MO2-ntn1a	angiogenesis disrupted, abnormal trunk vasculature lacks all parts of type parachordal vessel, abnormal