FIGURE
            Fig. 10
- ID
 - ZDB-FIG-141007-100
 - Publication
 - Li et al., 2014 - Overexpression of miR-19b Impairs Cardiac Development in Zebrafish by Targeting ctnnb1
 - Other Figures
 - All Figure Page
 - Back to All Figure Page
 
                
                    
                        Fig. 10
                    
                    
                
                
            
        
        
    
        
            
            
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 MiR-19b directly targets ctnnb1. A representative image of ctnb1 expression in 48 hpf embryos determined by in situ hybridization is shown (A). The expression in the miR-19b mimic-injected embryos was decreased when compared with WT and NC-injected embryos. Similarly, real-time PCR showed the expression level of ctnnb1 at 24 and 48 hpf was significantly downregulated in the mir-19b mimic-injected embryos. A schematic of the luciferase vector-expressing ctnnb1 32UTR construct is shown (C). Luciferase assay with ctnnb1 32UTR of WT, NC and miR-19b mimic-injected embryos is presented (D). ** P < 0.01; *** P < 0.001.  | 
    
                
                    
                        Expression Data
                    
                    
                
                
            
        
        
    
        
            
            
    | Gene: | |
|---|---|
| Fish: | |
| Anatomical Term: | |
| Stage: | Long-pec | 
                
                    
                        Expression Detail
                    
                    
                
                
            
        
        
    
        
            
                
            
        
    
    
    
                
                    
                        Antibody Labeling
                    
                    
                
                
            
        
        
    
        
            
                
            
        
    
    
    
                
                    
                        Phenotype Data
                    
                    
                
                
            
        
        
    
        
            
            
            
            
    
    
                
                    
                        Phenotype Detail
                    
                    
                
                
            
        
        
    
        
            
                
            
        
    
    
    
                
                    
                        Acknowledgments
                    
                    
                
                
            
        
        
    
        
            
            
                
                    
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      Full text @ Cell Physiol. Biochem.