FIGURE
            Fig. 6
- ID
 - ZDB-FIG-160308-17
 - Publication
 - Ponomareva et al., 2016 - Charcot-Marie-Tooth 2b associated Rab7 mutations cause axon growth and guidance defects during vertebrate sensory neuron development
 - Other Figures
 - All Figure Page
 - Back to All Figure Page
 
                
                    
                        Fig. 6
                    
                    
                
                
            
        
        
    
        
            
            
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 CMT2b Rab7 mutants do not cause increased cell death at 3 dpf. a-b Lateral views of 3 dpf wildtype (a) and CMT2b L129F (b) larvae labeled with anti-HuC/D in brown. Arrows indicate RB cell bodies. c Quantification of cell body number in five segments beginning at end of the yolk extension. CMT2b L129F is significantly greater than wildtype, *p = 0.02, student’s t-test. N = 20 embryos for each group  | 
    
                
                    
                        Expression Data
                    
                    
                
                
            
        
        
    
        
            
            
            
            
    
    
                
                    
                        Expression Detail
                    
                    
                
                
            
        
        
    
        
            
                
            
        
    
    
    
                
                    
                        Antibody Labeling
                    
                    
                
                
            
        
        
    
        
            
                
            
        
    
    
    
                
                    
                        Phenotype Data
                    
                    
                
                
            
        
        
    
        
            
            
  
    | Fish: | |
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| Observed In: | |
| Stage: | Protruding-mouth | 
                
                    
                        Phenotype Detail
                    
                    
                
                
            
        
        
    
        
            
                
            
        
    
    
    
                
                    
                        Acknowledgments
                    
                    
                
                
            
        
        
    
        
            
            
                
                    
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      Full text @ Neural Dev.