Phenotype
|
Fish
|
Conditions
|
Figures
|
heart rudiment morphology, abnormal
|
WT + MO1-gata5
|
standard conditions
|
Fig. 2
from Hempel et al., 2017
|
heart physical object quality, abnormal
|
WT + MO1-gata5
|
standard conditions
|
Fig. 1
from Holtzinger et al., 2007
|
heart cardiac muscle cell decreased amount, abnormal
|
WT + MO1-gata5
|
standard conditions
|
Fig. 5
from Jia et al., 2007
|
heart tube malformed, abnormal
|
WT + MO1-gata5
|
standard conditions
|
Fig. 4 ,
Fig. 5
from Peterkin et al., 2007
|
anterior lateral plate mesoderm nkx2.5 expression decreased amount, abnormal
|
WT + MO1-gata5
|
standard conditions
|
Fig. 4
from Wen et al., 2017
|
heart morphology, abnormal
|
WT + MO1-gata5
|
standard conditions
|
Fig. 4
from Wen et al., 2017
|
heart primordium left side unfused from heart primordium right side, abnormal
|
WT + MO1-gata5
|
standard conditions
|
Fig. 1
from Holtzinger et al., 2007
|
anterior lateral plate mesoderm myocardial precursor myl7 expression decreased amount, abnormal
|
WT + MO1-gata5
|
standard conditions
|
Fig. 4
from Wen et al., 2017
|
heart bifurcated, abnormal
|
WT + MO1-gata5
|
standard conditions
|
Fig. 1
from Holtzinger et al., 2007
|
heart rudiment bifurcated, abnormal
|
WT + MO1-gata5
|
standard conditions
|
Fig. 2
from Hempel et al., 2017
|
heart nppa expression spatial pattern, abnormal
|
WT + MO1-gata5
|
standard conditions
|
Fig. 4
from Wen et al., 2017
|
embryonic heart tube development disrupted, abnormal
|
WT + MO1-gata5
|
standard conditions
|
Fig. 4 ,
Fig. 5
from Peterkin et al., 2007
|
heart decreased size, abnormal
|
WT + MO1-gata5
|
standard conditions
|
Fig. 5
from Jia et al., 2007
|
anterior lateral plate mesoderm myocardial precursor myh7 expression decreased amount, abnormal
|
WT + MO1-gata5
|
standard conditions
|
Fig. 4
from Wen et al., 2017
|
heart morphogenesis disrupted, abnormal
|
WT + MO1-gata5
|
standard conditions
|
Fig. 4 ,
Fig. 5
from Peterkin et al., 2007
|
heart nppa expression increased distribution, abnormal
|
WT + MO1-gata5
|
standard conditions
|
Fig. 4
from Wen et al., 2017
|
heart morphogenesis process quality, abnormal
|
knu9Tg + MO1-gata5 (AB)
|
standard conditions
|
Fig. 6
from Park et al., 2009
|
heart bifurcated, abnormal
|
knu9Tg + MO1-gata5 (AB)
|
standard conditions
|
Fig. 6
from Park et al., 2009
|
heart cardiac muscle cell decreased amount, abnormal
|
hey2m145/m145 + MO1-gata5
|
standard conditions
|
Fig. 5
from Jia et al., 2007
|
heart decreased size, abnormal
|
hey2m145/m145 + MO1-gata5
|
standard conditions
|
Fig. 5
from Jia et al., 2007
|
heart morphogenesis disrupted, abnormal
|
WT + MO1-gata5 + MO1-gata6
|
standard conditions
|
Fig. 4 ,
Fig. 5
from Peterkin et al., 2007
|
heart formation disrupted, abnormal
|
WT + MO1-gata5 + MO1-gata6
|
standard conditions
|
Fig. 3
from Peterkin et al., 2009
|
fin bud aplastic, abnormal
|
WT + MO1-gata5 + MO1-gata6
|
standard conditions
|
Fig. 6
from Peterkin et al., 2009
|
whole organism mir210 expression increased amount, abnormal
|
WT + MO1-gata5 + MO1-gata6
|
standard conditions
|
Fig. 2
from Jia et al., 2018
|
cardioblast migration to the midline involved in heart field formation disrupted, abnormal
|
WT + MO1-gata5 + MO1-gata6
|
standard conditions
|
Fig. 3
from Peterkin et al., 2009
|
myeloid cell differentiation decreased occurrence, abnormal
|
WT + MO1-gata5 + MO1-gata6
|
standard conditions
|
Fig. 3
from Jia et al., 2018
|
otic placode foxj1b expression decreased amount, abnormal
|
WT + MO1-gata5 + MO1-gata6
|
standard conditions
|
Fig. S3
from Jia et al., 2018
|
embryonic heart tube development disrupted, abnormal
|
WT + MO1-gata5 + MO1-gata6
|
standard conditions
|
Fig. 4 ,
Fig. 5
from Peterkin et al., 2007
|
yolk syncytial layer slc3a2a expression decreased amount, abnormal
|
WT + MO1-gata5 + MO1-gata6
|
standard conditions
|
Fig. S3
from Jia et al., 2018
|
granulocyte mpx expression decreased amount, abnormal
|
WT + MO1-gata5 + MO1-gata6
|
standard conditions
|
Fig. 3
from Jia et al., 2018
|
myeloid cell differentiation disrupted, abnormal
|
WT + MO1-gata5 + MO1-gata6
|
standard conditions
|
Fig. 1
from Peterkin et al., 2009
|
heart tube malformed, abnormal
|
WT + MO1-gata5 + MO1-gata6
|
standard conditions
|
Fig. 4 ,
Fig. 5
from Peterkin et al., 2007
|
heart morphogenesis disrupted, abnormal
|
WT + MO1-gata5 + MO1-smarcd3b
|
standard conditions
|
Fig. 1
from Lou et al., 2011
|
heart tube bifurcated, abnormal
|
WT + MO1-gata5 + MO1-smarcd3b
|
standard conditions
|
Fig. 1
from Lou et al., 2011
|
cardiac muscle cell decreased amount, abnormal
|
WT + MO1-gata5 + MO1-smarcd3b
|
standard conditions
|
Fig. 1
from Lou et al., 2011
|
cardiac muscle cell differentiation disrupted, abnormal
|
WT + MO1-gata5 + MO1-smarcd3b
|
standard conditions
|
Fig. 1
from Lou et al., 2011
|
cardiac muscle cell differentiation disrupted, abnormal
|
WT + MO1-gata5 + MO1-tbx5a
|
standard conditions
|
Fig. 1
from Lou et al., 2011
|
heart tube bifurcated, abnormal
|
WT + MO1-gata5 + MO1-tbx5a
|
standard conditions
|
Fig. 1
from Lou et al., 2011
|
heart morphogenesis disrupted, abnormal
|
WT + MO1-gata5 + MO1-tbx5a
|
standard conditions
|
Fig. 1
from Lou et al., 2011
|
cardiac muscle cell decreased amount, abnormal
|
WT + MO1-gata5 + MO1-tbx5a
|
standard conditions
|
Fig. 1
from Lou et al., 2011
|
heart morphogenesis disrupted, abnormal
|
WT + MO1-gata5 + MO3-gata4
|
standard conditions
|
Fig. 4 ,
Fig. 5
from Peterkin et al., 2007
|
heart tube malformed, abnormal
|
WT + MO1-gata5 + MO3-gata4
|
standard conditions
|
Fig. 4 ,
Fig. 5
from Peterkin et al., 2007
|
embryonic heart tube development disrupted, abnormal
|
WT + MO1-gata5 + MO3-gata4
|
standard conditions
|
Fig. 4 ,
Fig. 5
from Peterkin et al., 2007
|
otic placode foxj1b expression amount, ameliorated
|
mir210nju49/nju49 + MO1-gata5 + MO1-gata6
|
standard conditions
|
Fig. S3
from Jia et al., 2018
|
myeloid cell differentiation occurrence, ameliorated
|
mir210nju49/nju49 + MO1-gata5 + MO1-gata6
|
standard conditions
|
Fig. 3
from Jia et al., 2018
|
yolk syncytial layer slc3a2a expression amount, ameliorated
|
mir210nju49/nju49 + MO1-gata5 + MO1-gata6
|
standard conditions
|
Fig. S3
from Jia et al., 2018
|
granulocyte mpx expression amount, ameliorated
|
mir210nju49/nju49 + MO1-gata5 + MO1-gata6
|
standard conditions
|
Fig. 3
from Jia et al., 2018
|
heart morphogenesis arrested, abnormal
|
WT + MO1-gata5 + MO1-smarcd3b + MO1-tbx5a
|
standard conditions
|
Fig. 1
from Lou et al., 2011
|
cardiac muscle cell decreased amount, abnormal
|
WT + MO1-gata5 + MO1-smarcd3b + MO1-tbx5a
|
standard conditions
|
Fig. 1
from Lou et al., 2011
|
cardiac muscle cell differentiation disrupted, abnormal
|
WT + MO1-gata5 + MO1-smarcd3b + MO1-tbx5a
|
standard conditions
|
Fig. 1
from Lou et al., 2011
|
heart tube bifurcated, abnormal
|
WT + MO1-gata5 + MO1-smarcd3b + MO1-tbx5a
|
standard conditions
|
Fig. 1
from Lou et al., 2011
|
heart duplicated, abnormal
|
cy13Tg + MO1-gata5
|
standard conditions
|
Fig. 3
from Lin et al., 2013
|
lateral mesoderm EGFP expression decreased amount, abnormal
|
hsc70Tg + MO1-gata5 + MO1-gata6
|
standard conditions
|
Fig. 2
from Yuan et al., 2018
|
margin EGFP expression absent, abnormal
|
hsc70Tg + MO1-gata5 + MO1-gata6
|
standard conditions
|
Fig. 2
from Yuan et al., 2018
|
heart physical object quality, abnormal
|
twu34Tg + MO1-gata5 + MO1-gata6
|
standard conditions
|
Fig. 2
from Holtzinger et al., 2007
|
cardiac muscle cell decreased amount, abnormal
|
twu34Tg + MO1-gata5 + MO1-gata6
|
standard conditions
|
Fig. 2
from Holtzinger et al., 2007
|
heart morphogenesis disrupted, abnormal
|
twu34Tg + MO1-gata5 + MO1-smarcd3b
|
standard conditions
|
Fig. 1
from Lou et al., 2011
|
heart bifurcated, abnormal
|
twu34Tg + MO1-gata5 + MO1-smarcd3b
|
standard conditions
|
Fig. 1
from Lou et al., 2011
|
cardiac muscle cell differentiation disrupted, abnormal
|
twu34Tg + MO1-gata5 + MO1-smarcd3b
|
standard conditions
|
Fig. 1
from Lou et al., 2011
|
cardiac muscle cell differentiation disrupted, abnormal
|
twu34Tg + MO1-gata5 + MO1-tbx5a
|
standard conditions
|
Fig. 1
from Lou et al., 2011
|
heart morphogenesis disrupted, abnormal
|
twu34Tg + MO1-gata5 + MO1-tbx5a
|
standard conditions
|
Fig. 1
from Lou et al., 2011
|
heart bifurcated, abnormal
|
twu34Tg + MO1-gata5 + MO1-tbx5a
|
standard conditions
|
Fig. 1
from Lou et al., 2011
|
heart physical object quality, abnormal
|
twu34Tg + MO1-gata5 + MO2-gata4
|
standard conditions
|
Fig. 2
from Holtzinger et al., 2007
|
heart bifurcated, abnormal
|
twu34Tg + MO1-gata5 + MO2-gata4
|
standard conditions
|
Fig. 2
from Holtzinger et al., 2007
|
heart morphology, abnormal
|
el133Tg + MO1-gata5 + MO1-smarcd3b + MO1-tbx5a
|
standard conditions
|
Fig. S1
from Lou et al., 2011
|
pharyngeal musculature morphology, abnormal
|
el133Tg + MO1-gata5 + MO1-smarcd3b + MO1-tbx5a
|
standard conditions
|
Fig. S1
from Lou et al., 2011
|
whole organism EGFP expression absent, abnormal
|
knu9Tg + MO1-gata5 + MO1-gata6 + MO5-gata4 (AB)
|
standard conditions
|
Fig. 6
from Park et al., 2009
|
heart absent, abnormal
|
knu9Tg + MO1-gata5 + MO1-gata6 + MO5-gata4 (AB)
|
standard conditions
|
Fig. 6
from Park et al., 2009
|
endocardium aplastic, abnormal
|
s843Tg + MO1-gata5 + MO1-smarcd3b + MO1-tbx5a
|
standard conditions
|
Fig. S1
from Lou et al., 2011
|
heart morphogenesis disrupted, abnormal
|
twu34Tg + MO1-gata5 + MO1-smarcd3b + MO1-tbx5a
|
standard conditions
|
Fig. 1
from Lou et al., 2011
|
cardiac muscle cell differentiation disrupted, abnormal
|
twu34Tg + MO1-gata5 + MO1-smarcd3b + MO1-tbx5a
|
standard conditions
|
Fig. 1
from Lou et al., 2011
|
heart bifurcated, abnormal
|
twu34Tg + MO1-gata5 + MO1-smarcd3b + MO1-tbx5a
|
standard conditions
|
Fig. 1
from Lou et al., 2011
|