| Phenotype | Fish | Conditions | Figures | 
    
    
        
            | intersegmental vessel malformed, abnormal | y1Tg + MO1-prkcz | standard conditions | Fig. 6
                    
                    from Oubaha et al., 2012 
 | 
    
        
            | dorsal longitudinal anastomotic vessel aplastic, abnormal | y1Tg + MO1-prkcz | standard conditions | Fig. 6
                    
                    from Oubaha et al., 2012 
 | 
    
        
            | intersegmental vessel sprouting angiogenesis process quality, abnormal | y1Tg + MO1-prkcz | standard conditions | Fig. 6
                    
                    from Oubaha et al., 2012 
 | 
    
        
            | intersegmental vessel endothelial tip cell detached from dorsal aorta, abnormal | y1Tg + MO1-prkcz | standard conditions | Fig. 6
                    
                    from Oubaha et al., 2012 
 | 
    
        
            | intersegmental vessel has extra parts of type endothelial tip cell filopodium, abnormal | y1Tg + MO1-prkcz | standard conditions | Fig. 6
                    
                    from Oubaha et al., 2012 
 | 
    
        
            | nuclear migration disrupted, abnormal | knu3Tg/knu3Tg + MO1-prkci + MO1-prkcz | standard conditions | Fig. 6
                    
                    from Baye et al., 2007 
 | 
    
        
            | neuroblast nucleus position, abnormal | knu3Tg/knu3Tg + MO1-prkci + MO1-prkcz | standard conditions | Fig. 6
                    
                    from Baye et al., 2007 
 | 
    
        
            | neuroblast development disrupted, abnormal | knu3Tg/knu3Tg + MO1-prkci + MO1-prkcz | standard conditions | Fig. 6
                    
                    from Baye et al., 2007 
 | 
    
        
            | pigmented epithelial cell pigment granule mislocalised, abnormal | prkcim567/m567 + MO1-prkcz | standard conditions | Fig. 3
                    
                    from Cui et al., 2007 
 | 
    
        
            | retina structure, abnormal | prkcim567/m567 + MO1-prkcz | standard conditions | Fig. 3
                    
                    from Cui et al., 2007 
 | 
    
        
            | pigmented epithelial cell shape, abnormal | prkcim567/m567 + MO1-prkcz | standard conditions | Fig. 3
                    
                    from Cui et al., 2007 
 | 
    
        
            | determination of left/right asymmetry in lateral mesoderm decreased process quality, abnormal | AB/EKW + MO1-prkci + MO1-prkcz | standard conditions | Fig. 12  from Krock et al., 2014 
 | 
    
        
            | camera-type eye photoreceptor cell differentiation arrested, abnormal | AB/EKW + MO1-prkci + MO1-prkcz | standard conditions | Fig. 8  from Krock et al., 2014 
 | 
    
        
            | retina lacks all parts of type retinal cone cell, abnormal | AB/EKW + MO1-prkci + MO1-prkcz | standard conditions | Fig. 8  from Krock et al., 2014 
 | 
    
        
            | camera-type eye photoreceptor cell differentiation decreased occurrence, abnormal | AB/EKW + MO1-prkci + MO1-prkcz | standard conditions | Fig. 9  from Krock et al., 2014 
 | 
    
        
            | retina has fewer parts of type retinal rod cell, abnormal | AB/EKW + MO1-prkci + MO1-prkcz | standard conditions | Fig. 9  from Krock et al., 2014 
 | 
    
        
            | heart edematous, abnormal | TU + MO1-prkci + MO1-prkcz | standard conditions | Fig. 6  from Gerlach et al., 2014 
 | 
    
        
            | pronephric proximal straight tubule cell differentiation involved in pronephros development process quality, abnormal | TU + MO1-prkci + MO1-prkcz | standard conditions | Fig. 8  from Gerlach et al., 2014 
 | 
    
        
            | pronephric proximal convoluted tubule malformed, abnormal | TU + MO1-prkci + MO1-prkcz | standard conditions | Fig. 5  from Gerlach et al., 2014 
 | 
    
        
            | pronephros pronephric nephron tubule epithelial cell differentiation process quality, abnormal | TU + MO1-prkci + MO1-prkcz | standard conditions | Fig. 7  from Gerlach et al., 2014 
 | 
    
        
            | pronephric proximal convoluted tubule cell differentiation involved in pronephros development process quality, abnormal | TU + MO1-prkci + MO1-prkcz | standard conditions | Fig. 8  , 
                    
                        Fig. 9  from Gerlach et al., 2014 
 | 
    
        
            | pronephric proximal convoluted tubule endocytosis decreased occurrence, abnormal | TU + MO1-prkci + MO1-prkcz | standard conditions | Fig. 6  from Gerlach et al., 2014 
 | 
    
        
            | renal filtration decreased occurrence, abnormal | TU + MO1-prkci + MO1-prkcz | standard conditions | Fig. 6  from Gerlach et al., 2014 
 | 
    
        
            | pronephric proximal convoluted tubule apoptotic, abnormal | TU + MO1-prkci + MO1-prkcz | standard conditions | Fig. 5  from Gerlach et al., 2014 
 | 
    
        
            | pronephric proximal convoluted tubule apoptotic process increased occurrence, abnormal | TU + MO1-prkci + MO1-prkcz | standard conditions | Fig. 5  from Gerlach et al., 2014 
 | 
    
        
            | pronephric glomerulus development process quality, abnormal | TU + MO1-prkci + MO1-prkcz | standard conditions | Fig. 7  from Gerlach et al., 2014 
 | 
    
        
            | pronephric proximal tubule morphogenesis decreased process quality, abnormal | TU + MO1-prkci + MO1-prkcz | standard conditions | Fig. 5  from Gerlach et al., 2014 
 | 
    
        
            | pronephric tubule establishment of epithelial cell apical/basal polarity decreased occurrence, abnormal | TU + MO1-prkci + MO1-prkcz | standard conditions | Fig. 3  from Gerlach et al., 2014 
 | 
    
        
            | pigmented epithelial cell shape, abnormal | WT + MO1-prkci + MO1-prkcz | standard conditions | Fig. 2
                    
                    from Cui et al., 2007 
 | 
    
        
            | pigmented epithelial cell pigment granule mislocalised, abnormal | WT + MO1-prkci + MO1-prkcz | standard conditions | Fig. 2
                    
                    from Cui et al., 2007 
 | 
    
        
            | mitotic cell cycle process quality, abnormal | WT + MO1-prkci + MO1-prkcz | standard conditions | Fig. 3
                    
                    from Cui et al., 2007 
 | 
    
        
            | retinal ganglion cell layer disorganized, abnormal | knu3Tg + MO1-prkci + MO1-prkcz | standard conditions | Fig. 3
                    
                    from Cui et al., 2007 
 | 
    
        
            | cell migration process quality, abnormal | knu3Tg + MO1-prkci + MO1-prkcz | standard conditions | Fig. 9
                    
                    from Cui et al., 2007 
 | 
    
        
            | retina cellular quality, abnormal | knu3Tg + MO1-prkci + MO1-prkcz | standard conditions | Fig. 9
                    
                    from Cui et al., 2007 
 | 
    
        
            | amacrine cell spatial pattern, abnormal | knu3Tg + MO1-prkci + MO1-prkcz | standard conditions | Fig. 3
                    
                    from Cui et al., 2007 
 | 
    
        
            | pronephros establishment of epithelial cell apical/basal polarity decreased occurrence, abnormal | nz1Tg + MO1-prkci + MO1-prkcz | standard conditions | Fig. 4  from Gerlach et al., 2014 
 | 
    
        
            | pronephric nephron tubule morphogenesis decreased process quality, abnormal | nz1Tg + MO1-prkci + MO1-prkcz | standard conditions | Fig. 4  from Gerlach et al., 2014 
 | 
    
        
            | retina neuroepithelial cell positional polarity, abnormal | kca66Tg; kca6Tg + MO1-prkci + MO1-prkcz (AB) | standard conditions | Fig. 3
                    
                    from Cui et al., 2007 
 |